B (Bruxism) Splints


The B Splint is useful for rapid harmonization of occluso-muscle disorders. Its effectiveness is due to decreased muscle activity of the Lateral Pterygoids, Medial Pterygoids, Masseters, and Temporalis muscles created by malocclusion and/or parafunction (bruxism). Clinical EMG studies consistently show up to an 80% reduction in elevator muscle activity due to clenching.

Design of B Splint:

It is designed on the same principle as the classic anterior deprogrammer but can be used for an indefinite period of time when managed properly. Proper usage will not result in undesirable tooth movement.


Material: Biocryl  “blank”  (Great Lakes Orthodontics)
1mm thick  for short term usage
2 mm thick for long term usage

For daytime use:

1.    A single appliance is fabricated on either the upper or lower arch. A Biocryl “blank” sheet of 1 ½ or 2 mm thickness is heated and adapted to an unmounted cast using the Biostar(Great Lakes Orthodontics) positive air vacuum system. Block out any gross undercuts on the casts before processing.
 
2.    Orthodontic acrylic is mixed to a doughy stage and luted with liquid monomer to the incisor surface of the blank appliance. It is hand formed to create a platform that is thick enough to disclude all teeth but the opposing central incisors. This is determined by observation in the mouth on closing and recorded on a communication slip to the laboratory (sample provided).

The platform should be 6-8 mm wide or approximately half the width of the two opposing central incisors combined. It should be long enough to touch when the mandible is in the fully seated joint position as well as extended on a smooth horizontal surface (labially on a upper appliance/lingually on a lower appliance) to stay in contact when the mandibular incisors protrude out to an edge to edge position with the maxillary incisors.


It is very important that the platform be level with the benchtop and thus create a perpendicular contacting surface for the opposing teeth. It is critical that no distalizing forces to the mandible be present when the appliance is in place.
The orthodontic acrylic addition is cured in a pressure pot for 10 minutes.

For nighttime use:

When sleeping we want the patient wearing a “Dual Appliance”, upper and lower together. The benefit of this approach is to cover all teeth at night, thus the patient is wearing muscle deprogramming retainers. This prevents tooth movement/bite changes. This approach also:

  • prevents the patient from creating grooves in the B Splint from grinding at night
  • levels the incisal edge plane to prevent irregular tooth heights that can create problems of sore teeth
  • covers the two incisors that occlude against the B Splint that sometimes become sore


This second appliance opposing the B Splint should create a perfectly level and smooth surface for the patient to touch and function (or parafunction) against. 

Design of opposing appliance:

A second Biocryl “blank” (1 ½  or 2 mm sheet)  is fabricated on an unmounted cast of the opposite arch (upper or lower).
This will often have a thin addition of orthodontic acrylic, luted with monomer, over the incisors, to create a level incisal edge.
We call this a “runner bar”.
This addition of orthodontic acrylic is cured in a pressure pot for 10 minutes.

Instructions to the patient:

  • Wear only the B Splint during the day.
  • Take it out during meals which helps not only with eating but also to prevent any tooth movement of the non-contacting teeth of the opposing arch.
  • Wear both appliances at night.
  • Brush appliances daily with dentrifice.
  • If wearing the appliance(s) cause any increased discomfort... take it out and call the office to be evaluated.


Note: This approach can be used for an indefinite period of time with no negative effects.

We typically find that occluso-muscle symptoms can be resolved within 1-4 weeks. At that time the occlusion is evaluated for any needed corrections and the patient will discontinue use of the appliances… unless, parafunction continues at night. If this is the case, even if the occlusion is corrected, then nighttime use of the dual appliances is indicated for an indefinite period of time.

Patients who may be on a “holding pattern” for occlusal/restorative therapy may use the B Splint dual appliances every night, and the daytime single appliance as needed…for as long as needed.

12 comments (Add your own)

1. Raymond Kessler wrote:
Can these splints be used for muscle deprogramming on a patient with severe, flattened occlusion that needs full mouth rehab. Any new thoughts on opening the bite on these patients?

June 7, 2007 @ 7:42 PM

2. Jim Boyd, DDS wrote:
The photo above reveals two issues *critical* to the success of this type of device.

First, the worn tip of the lower canine confirms that the contralateral lateral pterygoid has been contracting with habitual intensity during sleep (there's no other way for the opposing canines to be approximated). Therefore, it may be assumed that the ipsilateral IHLP may have the same ability, which leads to the second issue.

The pictured Discluding Element (DE) does not extend beyond the edges of the upper incisors (it probably should), because if *both* IHLPs contract simultaneously during parafunctional activity, then the lower incisors can "fall up" off the end of the DE. The resulting protrusive clenching and sudden onset of new joint-related symptoms can be quite alaming, and may lead the practitioner to abruptly discontinue use of the device, assuming that there must be some previously unappreciated joint condition that is contra-indicated for a B-splint (or NTI...same therapeutic protocol, different means of retention).

This is a perfect example of how the opinion of these types of devices are contraindicated for certain joint conditions develops. It's not the design of the device, but the adherence to protocol that matters.

June 20, 2007 @ 9:40 AM

3. Charles A. Schultz,DMD wrote:
I would like to comment on an excellent lecture given by Dr. Wilkerson at the FNDC June 14,2007. In particular, an anecdote about a hygienist and her well used toothbrush. Teeth out of occlusion with root recession, apparently from brushing. Given the relatively recent work of Robert Vanarsdall at the University of Pennsylvania on anterior-posterior head films as periodontal risk markers, one becomes curious as to her maxillary-mandibular width ratio.
She may indeed brush a lot, being exceedingly prone to periodontal problems.

June 25, 2007 @ 12:18 PM

4. Charles A. Schultz,DMD wrote:

June 25, 2007 @ 12:25 PM

5. scott ma, dds wrote:
Dear colleagues:
The NTI, B splint, and many more of these devices which I called anterior stopper, all work to seat condylar in cr position and provide anterior guidance to disclude posterior teeth. I use self-curing acrylics tom fabricate the device. The important thing is I use articulating paper to mark the contact between teeth and anterior stopper. Keep on grinding the mark until you get stable occlusal contact between front teeth. I truly believe Dawson's bimanual manipulation technique can seat tmjs to musculoskeletally stable position, years of successful tmd treatment substantiayes the belief. When cr contact is marked by ariculating paper, you need to use different color to mark the contact made by patient volunteer contact, ie. without operator's manipulation.Two different color marks should overlap. If not, you need to meticulously grind the mark until cr position can be maintained by patient's self closure. In the beginning, you uaually see the pateint's self closure mark is in front of cr mark. I rarely see the self-closure mark behind cr mark. Once cr position is maintained, you proceed to establish anterior guidance.
respectfully
scott ma, dds

November 12, 2007 @ 9:41 PM

6. bruno giglio wrote:
how do you prevent posterior disclusion of a patient who is in need of a b splint who has upper CD complete lower dentition.

bruno

January 25, 2008 @ 12:21 PM

7. Larry Gottesman wrote:
I am currently undergoing a rather extensive literary review concerning the function of mechanoreceptors in anterior and posterior teeth and their role in neuromuscular coordination.

It seems that there is a misinterpretation as to the effects of anterior tooth stimulation and the jaw opening reflex.

Firstly, the jaw opening reflex refers specifically to the transient reflex exchanges between muscle groups as influenced and modulated by tooth contact in the anterior region, the central incisors having a dominant role in only an inhibitory contribution over the elevator muscles. During these brief reflex arcs. The information related to this reflex is mostly predicated on animal species with some recent human studies. The actual masticatory reflex is considered to be under the auspices of a central pattern generator (CPG) as a means of regulating or coordinating multiple tasks between several systems as an automatic phenomenon that can be modulated by various mechanoreceptive or cognitive stimuli.

Secondly, this reflex is signaled by activation of high threshold, rapidly adapting mechanoreceptors responding to directional tensive stimuli. They respond more to high phasic forces in order to rapidly unload the teeth as a protective mechanism.

Therefore, horizontal forces trigger the receptors located closer to the fulcrum area of the PDL. The low threshold, slowly adapting receptors are far more abundant and located in the apical region of anterior teeth. They respond to both phasic and sustained stimuli, however, their stimulation is excitatory in influencing muscle closure activation.

Thirdly, these excitatory and inhibitory reflexes occur in milliseconds with respect to the activation of the jaw closure inhibitory reflex. Subsequent to the inhibitory phase, there is a reinstitution of the excitatory mechanism.

Fourthly, molars have mostly excitatory jaw closure mechanoreceptor initiative and have a large predominance of low threshold mechanoreceptors responding mostly to axial compressive stimuli.

The abundance of tooth receptors descend from centrals to molars.

In light of this information, the NTI appliance or any appliance with a mini-anterior platform, which tends to axialize forces over the over lower centrals (also involved in the same reflex response as upper central incisors)may actually initiate some excitatory activitity of the elevator group, but without co-activation of the molar mechanoreceptors because they are out of occlusion. Aditionally, to my knowledge, there have been no studies on anterior deprogrammers, like the NTI, which isolate localization over the 2-4 anterior teeth and their opposing counterparts and the sustained effect load has on anterior mechanoreceptor activation. I think this makes the B splint a better choice because it does not load the anterior teeth selectively especially when used with a lower runner).

The NTI may actually work becauase it initiates an excitatory effect over closure muscles, like the Kois appliance, and allows the condyles to seat without interference from molars or their mechanoreceptors. In addition, the Kois appliance has an added benefit (in my opinion) of activating palatal soft tissue (which also contains mechanoreceptors) as a stimuli for decreasing muscle closure excitation.

I would also like to comment on appliances and intraarticular derangements. If there has been loss of normal integrity of the condyle disk assembly, whether congenital or traumatic, this is an internal derangement! It seems we save the use of this term for painful intraarticular sequelae. I think what we need is a classification of derangements to include the painful and nonpainful joints along with the other obvious parameters.

With respect to treating a painful intraarticular problem, if an anterior minimal incisal table appliance won't help, don't expect a splint with universal contacts to provide a greater benefit because it coactivates molar mechanoreceptors.

Finally, with respect to sleep bruxism, I have found that less damage occurs with an appliance similar to the B splint and the Kois as a hybrid between the two. Cuspid protection does not apply to sleep bruxism. Bruxism is not under the control of a conscious state. Most sleep bruxers continue to brux even in the appliance.

Thanks for the opportunity to share my knowledge with the Dawson folks...say HI to Pete and the rest of the gang for me.

Sincerely,

Larry Gottesman, DDS

March 5, 2008 @ 7:05 PM

8. Jim Boyd, DDS wrote:
There are numerous studies which isolate localization over the incisors and the sustained effect load has on anterior mechanoreceptor activation. The results are always the same. Maximal intensity of elevator contraction during molar contact is >3x that of incisor-only contact.

During nocturnal parafunction, there is no "reflex" to be concerned with. If "functional" reflexes were in play, there'd be no pathologic parafunction in the first place.

The provision of a "B-splint" is no different than an NTI in efficacy (so long as the Discluding Element is modified to control excessive protrusion, as the photo in this thread *does not*). The only drawback of the B-splint is the plastic that covers the posterior occlusal surfaces. In the presence of internal derangements, posterior freeway space must be minimal, and the occlusal plastic may prevent optimal therapy. (there is no validity to the notion that permanent posterior extrusion can happen during sleep).

As for the Kois Deprogrammer, its design is NOT intended for extended nocturnal use, unless it's Discluding Element is extended labially to accommodate for protrusive parafunction. Additionally, the palatal acrylic must be *tooth borne* by the anterior's cingulums, and not compress into the palatal mucosa.

Plastic doesn't help patients. Knowledge of the nature of parafunctional activity and how to control it, does.

-Jim Boyd, DDS

March 24, 2008 @ 12:30 AM

9. Larry Gottesman, DDS wrote:
Dear Dr. Boyd:

I would appreciate the names of the articles which localize stimuli over the anterior teeth in a sustained fashion. Thus far, I have been unable to identify the articles you tout as being supportive of your approach, although that is not to discount the efficacy of your treatment and the results. I suspect that the articles by many researchers out of Australia, UK and Sweden are among the group of authors whose work you refer to. I am not convinced that these works offer the information we seek especially during the conscious state parafunction.

Additionally, the parafunction described as "sleep bruxism" may be part of a reflex or central pattern generator that is very poorly understood and controlled, but obviously under the control of the voluntary state. Our problem is that sleep bruxism is destructive. However, the current studies related to this form of sleep disturbance do not concentrate on the effects of specific appliance design and outcome. I think this might be important to know in control of this form of parafunction and where our efficacy may reside. I suspect it will be quite some time before this information will be available with the parameters necessary to correctly assess and treat the disease states or not treat certain disease expressions because it is inappropriate.
Right now, much of what we do as treating desntists is to control the destructive capabilities of the violent forces generated by sleep bruxism.

While I would agree that understanding the nature of the disease is important, the modality I choose as my primary form of conservative therapy is plastic. Knowing what to do with the plastic is important! The freeway space may be critical if you look at it from that perspective, however, from a different point of view the condylar locus and force application to hard and soft tissues is probably a better way to visualize the pathophysiology and results. We often do this empirically by altering vertical dimension. The load locus is critical in some patients considering innervation of the supporting structures and disk.

This area of focal condylar position has received very little play and discussion within studies and the literature except from an empirical/anecdotal perspective and to identify CR. However, the relationship between the load locus and soft and hard tissues can be altered on the axis. This may be a crucial element in understanding some internal derangements amenable to conservative therapy. I have seen many cases needing more freeway space rather than less. I have no problem in helping a patient to feel better...I am just looking to understand the beast I treat better. I question everything because it leads me to ask better questions and gather more knowledge.

Thanks,

Larry Gottesman, DDS

March 25, 2008 @ 4:10 PM

10. Larry Gottesman, DDS wrote:
Jim: I meant to state that the sleep bruxism is not under control of the voluntary state!

March 25, 2008 @ 4:13 PM

11. Timothy Test DMD wrote:
The splint shown here is indicated to be a "B" splint...

What exactly is the origin of that name???

May 9, 2008 @ 8:54 PM

12. Elsa Wittbold, DDS wrote:
I don't understand "distalizing forces. " Could you please give an example of what would create a distalizing force.

Also, I am curious as to the emphasis on making the platform level with the bench top. Wouldn't we ideally want the platform to reflect the protrusive angle?

November 17, 2008 @ 9:41 AM

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